by New research published in the journal eGastroenterology sheds light on the hidden health risks of fine air particles, specifically those less than 2.5 micrometers in diameter (PM2.5), which are a major component of air pollution. These particles, when inhaled, can penetrate deep into the lungs and even enter the bloodstream, leading to various health concerns. Recent studies suggest that PM2.5 exposure can also damage the digestive system, affecting organs such as the liver, pancreas, and intestines.
The research focuses on the ways PM2.5 exposure triggers stress responses within the digestive system’s cells. These responses involve specialized cellular structures, including the endoplasmic reticulum (ER), mitochondria, and lysosomes. Disruptions to these organelles caused by PM2.5 can result in a chain reaction within the cells, leading to inflammation and other harmful effects.
The liver, a vital organ for detoxification and metabolism, is particularly susceptible to PM2.5 damage. Studies have shown that PM2.5 exposure can cause a cascade of issues within the liver, including inflammation, stress responses, and damage to organelles. These effects can contribute to the development of non-alcoholic fatty liver disease (NASH) and type 2 diabetes.
PM2.5’s impact on the digestive system does not end with the liver. The pancreas and intestines are also at risk. Research links PM2.5 exposure to an increased risk of pancreatic impairment in people with diabetes, as well as damage to intestinal cells and an increase in their permeability. This heightened permeability can lead to a range of digestive issues.
The seemingly invisible PM2.5 particles in the air we breathe can have profound effects on our digestive system, including the liver, pancreas, and intestines. These effects can contribute to various health issues, making it essential to minimize exposure to air pollution whenever possible.
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1. Source: Coherent Market Insights, Public sources, Desk research
2. We have leveraged AI tools to mine information and compile it
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